Se debe examinar Nombre de la prueba. La presión ocular interior, Tonometría . La forma y el color del nervio óptico, Oftalmoscopia (examen del ojo dilatado). Confiabilidad de la campimetría manual por confrontación para detectar defectos de campos visuales en patologías neurológicas. Article (PDF. Aumento de la presión intraocular; Degeneración nervio óptico; Daño de fibras ) Glaucoma 2º: diabetes, traumatismo ocular, cirugía ocular Campimetría.
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The objective is to report a 33 year old female who came to the emergency room of Ophthalmology complaining of reduced visual acuity on the left eye, in a progressive and insidious way, about two years ago. In the ophthalmological examination, she presented dilated tortuous vessels in her left bulbar conjunctiva, very high intraocular pressure and increased cupping of the optic disc.
CT scans of the skull and orbits, and ultrasonography of the eyeball and doppler of the ophthalmic artery and the supra-orbital veins had no abnormalities. Thus, it was suggested the possibility of idiopathic elevated episcleral venous pressure, an exclusion diagnosis, since intra-cranial and intraorbital pathologies were excluded.
The patient was treated medically with hypotensive eyedrops, with significant reduction of intraocular pressure on the left eye, but not enough, evolving to trabeculectomy. Glaucoma, Open-angle; Intraocular pressure; Episcleral venous pressure; Ultrasonography, doppler; Case reports. The intraocular pressure IOP depends on the rate of production of aqueous humour, the ease of its drainage and episcleral venous pressure 1. Most cases of dilated episcleral veins with increased IOP can be attributed to carotid-cavernous fistula, cavernous sinus thrombosis, dural arterio-venous shunt, superior vena cava syndrome, Sturge-Weber syndrome, dysthyroid orbitopathy, obstructive orbital injuries, or orbital varices 2.
There are rare cases of open-angle glaucoma and dilated episcleral veins without an apparent cause. This condition is known as idiopathic episcleral venous hypertension 1, The aim of this paper is to report a case of unilateral idiopathic episcleral venous hypertension in a young woman and to describe the diagnostic procedure for this relatively rare condition.
Cinco pruebas comunes para el glaucoma
She reported mild hyperaemia in the left eye starting eight years earlier. She was overweight with a history of dyslipidemia and was seeing an endocrinologist.
The patient was using no ocular medication and had no relevant ocular or family history. Extrinsic ocular motility was preserved. No proptosis of chemosis were observed, and retropulsion was normal. The patient had a reduced direct photomotor reflex and a prominent relative afferent defect in the LE.
Biomicroscopy of the LE showed mild dilation of episcleral vessels Figure 1clear cornea, deep and quiet anterior chamber, normal iris and clear lens. Biomicroscopy of the RE was normal. Fundus examination was normal in the RE, and oculae LE had a pale optic disc, with total excavation and discrete retinal venous dilation Figure 1. No carotid or orbital murmurs were found.
No relevant neurologic findings. Perimetry was not performed in the LE due to loss of central fixation. Fluorescein angiography was normal in the RE, and the LE showed a slight delay in venous filling in the region near the superior temporal arcade and dilation of perifoveal capillaries.
Computed tomography of the brain and orbits was within normal limits, as well as Doppler ultrasound of the eyeballs Figure 2. Laboratory tests thyroid hormones, rheumatologic tests, coagulation, serology for infectious diseases showed no relevant findings.
Given the very high IOP values in caampimetria LE, we opted for medical treatment with hypotensive eyedrops timolol maleate, brimonidine tartrate, dorzolamide hydrochloride and travoprost.
.: Portal da SBO – Sociedade Brasileira de Oftalmologia :.
The IOP was significantly reduced to 20mmHg, campimetris average, as was the retinal venous dilation Figure 1. However, due to the risk of glaucomatous damage, we decided to proceed with trabeculectomy. Arterio-venous fistula is the most frequent cause of ocular hyperaemia with elevated IOP, due to episcleral venous hypertension 8,9. The ocular hyperaemia results from dilated and arterialized conjunctival vessels, which are different from those found in episcleral venous hypertension, which is characterised by dilated episcleral vessels 1.
Idiopathic episcleral venous hypertension is a relatively rare syndrome that progresses with ocular hyperaemia and elevated IOP.
Jorgensen and Guthoff, while studying 64 patients with glaucoma due to episcleral venous hypertension, diagnosed this syndrome in 10 patients 8. In another report of six patients with unilateral dilated episcleral veins and one patient with bilateral involvement, the IOP of affected eyes ranged from 21 to 44 mmHg 4.
In the six patients with unilateral involvement, the IOP was higher in those eyes with dilated conjunctival vessels. The episcleral venous pressure was measured and corresponded to twice the normal value in the affected eyes of these patients campimefria. There is some variability in the clinical presentation of episcleral venous hypertension. The onset of signs dilated episcleral vessels may vary, usually occurring between the third and fourth decades of life 3.
Dilated episcleral veins may occur unilaterally or bilaterally.
Of the 25 cases reviewed in the English literature, 13 involved predominantly the RE, 3 involved predominantly the LE, and 9 had bilateral involvement, although asymmetrical. The ratio of men to women was Gonioscopy typically shows an open angle, and blood may or may not be visible in Schlemm’s canal. Of the 25 cases reviewed in the English literature, 24 had an open angle, of which 13 had blood in Schlemm’s canal 1, The condition should be distinguished from other diseases, the most common of which is arteriovenous fistula, which can cause dilated episcleral vessels and elevated IOP.
Although the cause of this disorder remains unclear, a congenital abnormality in the vasculature and familial predisposition have been considered as possible causes 5. Episcleral venous hypertension is a diagnosis of exclusion that must be considered after intraorbital and intracranial conditions have been discarded.
Cinco pruebas comunes para el glaucoma | Glaucoma Research Foundation
Diagnosis is based on the presence of dilated episcleral vessels, elevated IOP, and open angle on gonioscopy. Blood in Schlemm’s canal is a general sign of episcleral venous hypertension, but it is not always present, as was the case with our patient and other cases in the literature 3. The diagnostic evaluation should include a complete eye examination and imaging.
Some reports suggest performing magnetic resonance and even arteriography as part of the diagnostic investigation. However, in this campimetriw, we chose to perform a occular tomography of the head and orbits and a Doppler ultrasound; as these tests showed no abnormalities, more invasive and complex diagnostic tests were deemed unnecessary. These imaging tests are important because they can exclude other conditions.
Older reports mention the use of the MRI in a few cases, and although this is a more sensitive test than CT, it yielded no major findings, with most cases showing no abnormalities 3.
Doppler ochlar is a non-invasive method for examining the orbital and ocular blood flow and, in patients with fistula, it typically shows retrograde blood flow and a dilated superior ophthalmic vein 1. However, because this is a rare condition, it is difficult to estimate the sensitivity of such tests. The diagnosis of glaucoma secondary to idiopathic episcleral venous hypertension is clinical.
Measuring episcleral venous pressure is unpractical, with no commercially-available instruments to this end 3. The treatment of ocuular due to idiopathic episcleral venous hypertension with elevated IOP is similar to primary open angle glaucoma 3.
When medical treatment is not campimetris, filtration surgery is the procedure of choice. Ocjlar report mentions a patient who underwent trabeculectomy with subsequent reduction of the IOP, although the episcleral vessels remained dilated 6.
Additionally, there are reports in the literature of choroidal effusion after filtration surgery, showing oculsr the procedure is not without risks or complications 3. It is necessary to raise awareness among ophthalmologists about idiopathic episcleral venous hypertension, so that patients with dilated episcleral veins can be managed appropriately given the risks posed by the condition.
In addition to medical treatment with eyedrops, such patients may require filtration surgery, and ophthalmologists should exercise caution in performing this procedure because of the risk of postoperative choroidal effusion.
Case Report Revista vol. Idiopathic dilated episcleral veins and increased intraocular pressure. Glaucoma associated with elevated episcleral venous p ressure. Idiopathic elevated episcleral venous pressure and open-angle glaucoma. Increased pressure of dilated episcleral veins with open-angle glaucoma without exophthalmos. Dilated episcleral vessels and open-angle glaucoma.
Unilateral open-angle glaucoma secondary to idiopathic dilated episcleral veins. Familial glaucoma associated with elevated episcleral venous pressure.
Jorgensen JS, Guthoff R. The ocular findings in carotid-cavernous fistula in a series of 17 cases. Revista Brasileira de Oftalmologia.
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